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Each model type captures different aspects of wound biology, and GHK-Cus performance has varied across models in ways that reflect these underlying biological differences

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95% CI, 5.9-21.4), at least 15% (OR = 8.1

Other agents, such as the peroxisome proliferator-activated receptor (PPAR) agonist Saroglitazar, the apoptosis signal-regulating kinase 1 (ASK1) inhibitor Selonsertib, and the C-C chemokine receptor types 2 and 5 (CCR2/CCR5) antagonist Cenicriviroc, have shown limited or inconsistent efficacy in clinical trials, hindering broader application (175177)