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[DOI] [PubMed] [Google Scholar] Blair P.A., et al

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doi: 10.1152/physrev.00031.2024

Under lipotoxic conditions, excessive peroxisomal substrate flux may itself become a source of oxidant stress, while inadequate catalase-dependent detoxification can facilitate ROS spillover toward mitochondria, thereby aggravating membrane depolarization, inflammasome signaling, and bioenergetic collapse ( PPAR- is particularly important in regulating the activity of the antioxidant enzymes catalase and HO-1

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