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Instead of blocking inflammatory cytokines after they are produced or suppressing the immune cells that produce them, thymosin alpha-1 strengthens the natural regulatory mechanisms that should be preventing the autoimmune response in the first place

doi.org/10.3389/fphar.2021.627533 Chang, C

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The mechanism appeared to involve NO-mediated vasodilation and enhanced endothelial repair rather than direct anticoagulant activity

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