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A variety of cellular stresses, including DNA damage can cause the induction of cellular senescence, which represents a multifaceted and highly heterogeneous cellular state characterized by irreversible growth arrest and importantly the secretion of a multitude of factors, including proteases and chemokines 2

In general, these findings illustrate complexity associated with the contribution of oxidative stress and apoptosis/angiogenesis-related genes in early pregnancy loss and recurrent pregnancy loss

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In head and neck squamous cell carcinoma, PD-L1 binds to ILF3 and IL2 to activate STAT3 for PD-L1 signaling to promote tumor progression [66]

It contains growth factors that stimulate tissue repair