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Its peptide structure closely resembles that of endogenous GLP-1, allowing it to interact with GLP-1 receptors in the body [4]

Improvement timing varies

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Ikrar T, Guo N, He K, Besnard A, Levinson S, Hill A, et al

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This is in line with the notion that the IVD is immune privileged due to its avascularity [30] and the expression of Fas ligand (FasL), which induces apoptosis of invading Fas-bearing T-cells [24, 31]